Conditions Seen

Helping You Manage Various Health Conditions

For nearly 30 years, Dr. David Marquis has assisted thousands of patients to find solutions to acute and chronic conditions without the use of or with minimal use of drugs or surgery. Through a metabolic and neurological approach to his patient’s health conditions, not only can the causes be determined, but the patient can learn how to manage their own health and often without medication. This is the underlying goal of care in Dr. Marquis’s office. Finding solutions and empowering patients to take their health back and become nondependent on the broken insurance-driven medical paradigm that plagues our country today.

When working with patients it is often identified that their real problems are the results of unrecognized neurometabolic issues. This means that they may have metabolic dysfunction such as blood sugar imbalances, autoimmune conditions, anemias, food sensitivities, latent infections, or simple nutritional imbalances which are complicating normal metabolism. Along with these metabolic issues, there are often neurological hurdles as well that lend themselves to the many avenues of brain-based therapies that Dr. Marquis has been trained in over the years.

Dr. Marquis has successfully helped patients navigate through a variety of neurological, metabolic, and chiropractic conditions including, but not limited to:

Successfully Treating Metabolic Conditions

Insulin Resistance and Diabetes

Insulin resistance and diabetes are more often than not the result of eating the wrong foods over time. Whether you have a poor diet or your body has autoimmune sensitivities to foods you include in your diet, the result is the same. Under ideal circumstances, the body is able to properly metabolize glucose at the cell level, creating the energy necessary for the brain and other organs to both function and repair themselves. Autoimmune issues and poor diet can both contribute to an environment wherein cells don’t metabolize glucose properly. This situation sets off a series of attempts by the brain to compensate (unsuccessfully) by sending out insulin and eventually increasing cortisol levels.

Failing to eliminate the causes that trigger this natural response in the body will lead to a cycle that has the potential of developing into type II diabetes, and other areas in your body begin functioning below desired levels. Fatigue, appetite, sleeping habits, stress management, and memory can all be affected by the increases in insulin and cortisol.

Through proper testing, we can determine the cause(s) of your symptoms that are pointing to insulin resistance and diabetes. We can then make diet and other lifestyle changes to help you better correct your glucose levels. Our patients don’t just strive to manage their condition when they do the work, we teach them to do they correct the underlying condition! This process leads to not only bringing blood sugar back into balance but it often leads the patient to a state of health they have not enjoyed in many years!

Check out our page on lab testing to get an idea of the process by which we start our evaluation and watch the video above to learn more about our approach to the correction of this major killer.

Stopping the Head Pain

The migraine headache is perhaps the best known special type of headache. A lot of things accompany this type of headache—all of them bad. Symptoms can include dizziness, visual problems, “spots” before the eyes, redness, swelling, tearing of the eyes, muscle contraction, irritability, nausea, vomiting, constipation, or diarrhea. These symptoms often arise before the headache hits. The headache itself may last for a few minutes to a few days, and the severity may range from minor discomfort to immobilizing agony. Migraine pain is most commonly felt in the temple, but it may be experienced anywhere in the head, face, and neck.

Another variety of headache, closely related to the migraine, is the cluster headache. Attacks come on abruptly with intense, throbbing pain arising high in the nostril and spreading to behind the eye on the same side. Sometimes, the forehead is also affected. The attacks tend to occur from once to several times daily in clusters lasting weeks, or even months. Without apparent reason, the cluster subsides as quickly as it began.

One more common type of headache is called a sinus headache. This frequently can present as atmospheric pressure changes occur if the individual has chronic inflammation in their sinuses cavities. This type of headache responds well to food sensitivity testing and dietary modification as well as in the moment of pain with laser therapies which can rapidly reduce the pressure in the sinuses.

One more that we see a lot of is the cervicogenic or “tension-type” headache which is caused by improper muscle firing patterns in the neck, head, and shoulders. This type is the most common cause of head pain and comprises a full 80% of the people who experience headaches. This is another type that responds fabulously to laser therapy as well as proper oxygenation and magnesium.

So what puts the ache in headache? The pain-sensitive structures of the head are the culprits. These are the arteries of the brain and skull, the tissues surrounding the head, the veins, the dura mater covering over the brain, and certain nerves called cranial nerves. When these parts are inflamed, stretched, pulled, or under pressure, any type of headache may be caused.

Migraine headaches can be classified into two types: classical and common. The classical migraine is a headache that follows an aura or some type of spontaneous events such as numbness or tingling. The aura may be flashes of light, squiggly lines, or a halo effect. The common migraine does not have an aura associated with it. Most people who suffer from migraines suffer from common migraines—usually at a 3:1 ratio.

Approximately 28 million Americans suffer from migraines, and millions go without treatment. Scientists once thought migraines were caused by abnormally dilated or enlarged blood vessels. Now, new imaging devices have allowed them to watch brains during migraine attacks, and scientists are discovering that sufferers have abnormally excitable neurons or brain nerve cells.

The latest migraine research has yielded a mechanism called cortical spreading depression, or CSD. Prior to the onset of pain in a migraine, researchers have observed a sudden burst of cortical activity that occurs most commonly in the occipital lobes (back part of the brain). The occipital lobe will increase in the frequency of firing, or have a burst of activity, and then there will be an episode of silence or depressed activity. The actual activity of the brain becomes depressed when compared to normal. The resulting pain comes from either the brain stem activation or from blood vessels inflamed by rapidly exchanging blood flow or both.

I take a different approach to the treatment and prevention of headaches and migraines. After a thorough neurological examination, I determine which part of the nervous system is not functioning properly, and if there is proper fuel delivery in the form of oxygen and glucose. In many headache and migraine patients, I may find a high mesencephalic output. There are three parts to the brain stem: top, middle, and lower. The mesencephalon is the top part of the brain stem. A high-output of the mesencephalon that is not dampened by the lower portions (the pons and medulla) will cause an increased pulse and heart rate, inability to sleep, or waking up from fitful sleep, urinary tract infections, increase warmth or sweating, and sensitivity to light. Often times, in these patients poor muscular firing patterns have developed that lead to chronic spasms in the involuntary muscles of the neck and shoulders. For these patients, utilizing laser therapies has been extremely successful at minimizing and sometimes removing one more trigger for their pain.

I think I must have had headaches all of my life, but the first time I remember asking a doctor about them was when I was 28. I got through them the next 35 years with over the counter drugs, and then I developed a bleeding ulcer and couldn’t take the O.T.C. meds any longer. 20 years ago I began getting headaches about 2-3 hours after I went to sleep, so I started on a quest to find an answer. I have done many protocols – infusions, hormone therapy, food allergies, and several different kinds of pillows. Recently my primary doctor sent me to Dr. Marquis. 3 weeks ago I started laser treatments, I have only taken 5 Maxalt in that time (I was taking 3 per day). Only 3 of the headaches in the past 3 weeks were at night. This has been the best 3 weeks of my life in 20 years!

Delphia R. Connella

There are metabolic triggers for head pain as well. A common one is the surprising array of food sensitivities. Many times, the culprit can be foods that are considered “healthy.” I have seen just as many headaches caused by dairy and bread as I have food dyes and flavorings. The best way to identify the individual trigger is through comprehensive metabolic blood testing. This is an invaluable tool that we use for most chronic conditions and are able to provide beneficial life-changing information once we gain the data. No matter what the condition, it is imperative that the doctor performs a thorough and comprehensive exam to determine the exact nature of the patient’s condition. If you would like to have more information or to set up a consultation and see how we can help you (or someone you know)

The functional medicine system of care has helped thousands of people across the country and is now available in the Central Coast.

Peripheral Neuropathy describes damage to the nervous system in your legs, feet, arms, or hands. This vast communications network transmits information from the brain and spinal cord (the central nervous system) to every other part of the body. Peripheral nerves also send sensory information back to the brain and spinal cord, such as a message that the feet are cold or a finger is burned. Damage to the peripheral nervous system interferes with these vital connections. Like a poor cell phone connection, peripheral neuropathy distorts and sometimes interrupts messages between the brain and usually the endings of nerves.

Patients with peripheral neuropathy experience a variety of symptoms such as chronic tingling, numbness, weakness, or even burning pain. They often find it difficult to walk or sense the type of surface under their feet. They often don’t even know if they have injured the affected area and are often miserable because of the chronic pain they experience.

Our approach is comprehensive in helping our patients with peripheral neuropathy. Not only do we treat the local area affected by using cutting edge therapy, addressing metabolic conditions (such as blood sugar problems) but we also treat areas in the brain responsible for receiving these messages from the body. Covering both the neurological and metabolic aspects of this disease is vital for any treatment to truly be successful. It is common that peripheral nerve damage will lead to functional changes in the brain! You MUST address all areas in order to get the BEST OUTCOMES.

Many people have seen images of the brain with our body superimposed over it displaying specific body parts and how they relate to the brain. There is a ‘road map’ in the brain of your entire body. Every body area (foot, hand, face, etc.) is represented in a part of the brain called the parietal lobe. If poor nerve signals come into the brain because of peripheral neuropathy, this can lead to problems in the brain itself. Allowed to progress and the brain actually changes over time and becomes less and less functional. This is termed neuroplasticity. This plasticity can work for us or against us depending on the information you provide the brain.

Although you might start out with peripheral neuropathy, the longer you have this condition the more likely you’ll experience issues higher up into areas of the brain. That is why damage to the peripheral nerves is just part of the story. And why treating the local area can lead to discouraging short-term results.

We Offer So Much More... a Unique Nondrug Approach

For peripheral neuropathy, our evaluation starts with a unique exam based on the Toronto Clinical Scoring System. Why? Because this exam scores the health or sickness or your nerves. In-depth laboratory analysis provides important details as to the CAUSE of your condition. Wouldn’t it be nice to objectively demonstrate where you are and how you are improving? Not just “I feel good today” but an exam that proves you are getting better. How is this accomplished? Through the unique combination of clinically proven therapies…


We use a combination of brain-based therapy (BBT), laser and infrared therapy, spinal decompression when necessary, whole body vibration, Exercise with Oxygen Therapy (EWOT), The ReBuilder, and other brain-based therapies (BBT) geared toward your specific condition to address peripheral neuropathy in a very dynamic way.


On top of the brain-based therapies (BBT), we also support the nervous system using advanced metabolic and nutritional protocols designed to give you every chance possible to feel better. If you want to restore normal nerve communication, function, and health it is best to start healing from the inside out!

No matter what the condition, it is imperative that we perform a thorough and comprehensive exam to determine the exact nature of the patient’s condition.

While searching for better ways to help my patients, I was fortunate to learn of Dr. Datis Kharrazian, a pioneer in natural health care and author of the best selling book “Why Do I Still Have Thyroid Symptoms When My Thyroid Tests Are Normal?“ I have spent a great deal of time studying his work and in fact learning directly from him for quite some time now. On this page, you will have the opportunity to learn about some fundamental aspects of thyroid function and dysfunction. I highly recommend reading Dr. Kharrazian's book as well.

Introduction to Functional Thyroid Disorders

Functional thyroid disorders are incredibly common and often overlooked in our current health care model. Most patients that have functional thyroid imbalances do not have primary thyroid imbalances. In fact, 5 out of every 6 thyroid patients are suffering from an undiagnosed auto-immune problem driving their thyroid problem.

Thyroid problems are often misdiagnosed and mistreated... and women are often affected the most. Over 80% of our patients who have a thyroid problem are not being treated in a manner that will provide them long-term solutions and many are experiencing their problems worsen because the root cause has been overlooked.

See if this sounds familiar? Your labs come back “normal” and yet you still feel tired, achy, cold, depressed, and mentally sluggish. I can’t tell you how many people have been told…”go home, get some rest, your blood work looks normal, you are probably just overworked.” Or even worse, you are put on thyroid hormone replacement and you still have all the symptoms of hypothyroidism but your lab tests show that your thyroid levels are good. And worst of all it’s difficult to get anyone to listen to you. So now what do you do? You go through your day with any one or more of these symptoms (some poor souls have them all!).

Book by Dr. Kharrazian

It doesn’t have to be that way. I have found that most patients will have inadequate labs (often only two tests done: TSH and T4) to determine if they have a thyroid problem. These two tests offer a simple screening for thyroid function but they certainly don’t provide the complete picture! Did you know there are over 20+ ways that your thyroid can go bad? Fundamentally, there are 6 major categories for thyroid dysfunction and 24 subsets off of these 6. And only one of those ways will respond to typical replacement therapy. So, why is everyone treated with hormone replacement if only 1 out of 6 will actually be resolved with the medication? Not everyone fits into the replacement model of treatment. Modern approaches allow us to determine your specific needs and then let the body correct itself with proper neurological and metabolic support. Don’t go on feeling frustrated. We will always listen to your concerns and help you chart a course to get you back on track. You need comprehensive blood work (see our Laboratory Analysis page) and functional neurological analysis will help us determine the shortest path to getting you better.

If you are ready to reserve an appointment, please call
(805) 481-3499. Below you will find some core information on thyroid and the physiology of the gland.

Thyroid Physiology Review

Once the thyroid is stimulated by thyroid-stimulating hormone (TSH) from the pituitary, it produces thyroxine (T4) and triiodothyronine (T3) by transporting iodine into the thyroid and by stimulating Thyroid Peroxidase Activity (TPO). TPO is involved in the formation of T4 and T3 as it catalyzes the oxidation of iodine using hydrogen peroxide. The thyroid will produce 94% of the available T4 and 7% of the available T3. As we know, T4 is inactive and T3 is an active thyroid hormone. Therefore, the majority of hormone production at the thyroid is inactive T4. Once the thyroid has produced T4, it is metabolized peripherally from the thyroid into combination T3 hormones by the enzyme 5′ deiodinase, mostly at the liver. Under normal circumstances, about 40% of the available T4 is converted into T3, 20% is converted into reverse T3 (rT3), which is irreversibly inactive, and 20% is converted into T3 sulfate (T3S) and triiodothyroacetic acid (T3AC). T3S and T3AC are inactive thyroid hormones until they circulate into the gastrointestinal tract and are acted upon by intestinal sulfatase into active T3. Gastrointestinal sulfatase activity is dependent upon a healthy gut microflora.

So with most of your thyroid hormone conversion occurring in the liver and gut, what happens if your liver function is compromised or your have an inflamed gut. Do you know anyone with either or both of those problems? The funny thing is (and it's not really funny) that most people end up being told they have 3 distinct problems and never realize that their GI problems and toxic liver and thyroid dysfunction are all part of the same disease. This is why so many people are suffering and being medicated but not finding a resolution to their complaints. Looking at thyroid dysfunction requires a comprehensive metabolic and neurological approach. In this way no stone is left unturned and although the process generally takes time patients do finally find the relief they have been seeking.

Understanding Thyroid Markers and Panels

TSH: thyroid stimulating hormone (TSH) is also called thyrotropin. The pituitary releases this hormone after the hypothalamus releases TRH (thyrotropin-releasing-hormone). This is the most common marker used to assess thyroid function and it is also the most sensitive. The TSH levels increase when the T4 levels drop, and the TSH falls when T4 levels increase. This is the only test performed in the traditional health care model as a means to screen the patient for thyroid disorders; this is because they are only concerned for screening the thyroid for hormone replacement and not optimal physiological function. A TSH test alone does not consider thyroid pituitary feedback loops, peripheral thyroid metabolism, or potential or active risk factors as identified by antibody testing. A high TSH with or without changes in T4 or T3 is diagnostic to determine hypothyroidism. If the thyroid is not making enough T4, the pituitary will pump out TSH to stimulate its production. A low TSH is used to determine hyperthyroid activity. If the thyroid is overactive such as in Grave’s disease, the antibodies bind to active thyrotropin (TSH) receptors on the thyroid cells and stimulate T4 production without the influence of TSH. Please note that some antibodies may inhibit thyroid function by inactivating instead of stimulating thyrotropin receptors. This is called an autoimmune hypothyroid. These patterns will demonstrate a hypothyroid pattern (elevated TSH) with elevated thyroid antibodies.

Laboratory Reference Range: 0.5 – 5.5 (varies from one lab to another)

Functional Reference Range: 1.5-3.5

Total Thyroxine (TT4): The TT4 test measures both bound and unbound thyroxine levels, therefore, it does not give the activity of T4 when measured alone. This test is best completed with a T3 uptake. The free thyroxine index (FT4) can be calculated by using the T3 uptake and demonstrating a level of T4 activity. Total T4 levels can be altered by many drugs.

Free Thyroxine Index: As stated earlier, the total thyroxine and T3 uptake must be used together to calculate the FT4. The index is measured by multiplying the TT4 levels by the T3 uptake levels. The result is the FT4 and it determines the amount of active T4 available. The impact of drugs, as will be discussed, will always impact T4 and resin T3 uptake levels in opposite directions due to its impact on binding sites. If the TT4 level is depressed, then the T3 uptake is high; if the TT4 is elevated, the resin uptake is low. Please note that even if you are taking drugs that may impact thyroid-binding, the free thyroxine index should be within the normal range if your thyroid is functioning normally.

Free Thyroxine (FT4): The free thyroxine test is used to measure the amount of free or active T4 in the blood. All the factors such as drugs and physical conditions that may impact the TT4 do not impact the FT4. The level of T4 in the blood is high with hyperthyroidism or low with hypothyroidism. Please note that even a high TSH with normal T4 is enough to diagnose hypothyroidism. A rare pattern is an elevated T4 without hyperthyroidism, which may be related to a hereditary condition of thyroid resistance. Elevated Free T4 may also be caused by patients taking heparin or by an acute illness that may briefly cause the binding protein levels to suddenly fall. If an illness becomes severe and chronic, it may decrease the FT4 levels but it is not a thyroid disease.

Resin T3 Uptake: The resin T3 uptake measures the amount of sites for active (unbound) T3 to bind on thyroxine-binding proteins. This test is performed by mixing the blood with radioactive thyroid hormones. These radioactive hormones then combine with binding sites on thyroxine-binding proteins. The blood is then exposed to a substance called a resin that will bind the unbound thyroid hormones, and measure for radioactivity. The result can be expressed as the percent of radioactivity found on the resin compared to the original radioactivity that was added. The more binding sites that are open on the proteins, the lower the resin-uptake result will be, and vice versa. For example, anything that reduces the binding sites, such as elevated testosterone or testosterone replacement therapy, causes a low T4 measurement because it leaves very few binding sites for any more thyroid hormone to bind to it. If T3 is added to the sample of the blood, little T3 will be bound. This pattern would have low TT4 levels and high resin T3 uptake levels. On the other hand, anything that raises the binding sites, such as estrogen or birth control pills, would cause a pattern of high TT4 and low T3 uptake.

Free Triiodothyronine (FT3): This test measures the free T3 hormone levels. This test is rarely completed in traditional endocrinology. It is typically only used in a situation where the patient has hyperthyroid, yet the FT4 levels are normal. However, the FT3 test is the best marker to see what amount of active thyroid hormones is available for the thyroid receptor sites.

Reverse T3 or (rT3): This test measures the amount of reverse T3 that is produced. The production of rT3 typically takes place in cases of extreme stress such as major trauma, surgery, or severe chronic stress. It appears that the increased production of reverse T3 is due to the inability to clear rT3, as well as from elevated cortisol.

Thyroid Antibodies: Thyroid autoantibodies indicate that the body’s immune system is attacking itself. Production of thyroid autoantibodies may create a hypothyroid or a hyperthyroid state. Some antibodies attach to the TSH receptors but do not cause a response. Therefore, the patient will complain of low thyroid symptoms, however, the serum TSH may not be altered. It is just not able to cause a cellular change. On the other hand, some antibodies will bind to the receptor sites and cause over-activation of the thyroid. This will present as elevated T4 levels, a low TSH, and elevated thyroid antibodies.

Low Thyroid Symptoms

  • Fatigue
  • Increase in weight gain even with low-calorie diet
  • Morning headaches that wears off as the day progresses
  • Depression
  • Constipation
  • Hypersensitivity to cold weather
  • Poor circulation and numbness in hands and feet
  • Muscle cramps while at rest
  • Catches colds and other viral/bacterial problems easily and has difficulty recovering
  • Wounds heal slowly
  • Excessive amount of sleep required to function properly
  • Chronic digestive problems (hypochlorhydria)
  • Itchy dry skin

Low Thyroid Signs

  • Dry or brittle hair
  • Hair falls out easily
  • Dry skin
  • Low axillary temperature (this may also be caused by an endocrine imbalance)
  • Edema, especially facial (myxedema)
  • Loss of outside portion of eyebrows

Drugs That Influence Thyroid Metabolism

Drugs That Decrease TSH Secretion:

  • Dopamine
  • Glucocorticoids
  • Octreotide

Drugs That Alter Thyroid Hormone Secretion by Decreasing Secretion:

  • Lithium
  • Iodide
  • Aminoglutethimide

Drugs That Alter Thyroid Hormone Secretion by Increasing Secretion:

  • Iodide
  • Amiodarone

Drugs That Decrease T4 Absorption:

  • Colestipol
  • Cholestyramine
  • Aluminum hydroxide
  • Ferrous sulfate
  • Sucralfate

Drugs That Alter T4 and T3 Transport in Serum by Increasing TBG Concentrations:

  • Estrogens
  • Tamoxifen
  • Heroin
  • Methadone
  • Mitotane
  • Fluorouracil

Drugs That Alter T4 and T3 Transport in Serum by Decreasing TBG Concentrations:

  • Androgens
  • Anabolic steroids
  • Slow-release nicotinic acid
  • Glucocorticoids

Drugs That Alter T4 and T3 Transport by Displacement Form Protein-Binding Sites:

  • Furosemide
  • Fanclofanac
  • Mefenamic acid
  • Salicylates

Drugs That Alter T4 and T3 Metabolism by Increasing Hepatic Metabolism:

  • Phenobarbital
  • Rifampin
  • Phenytoin
  • Carbamazepine

Drugs That Decrease T4 5′ Deiodinase Activity:

  • Propylthiouracil
  • Amiodarone
  • Beta-adrenargic antagonist drugs

Offering Professional Advice on Neurodegenerative Diseases


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Post-Concussive Syndrome


Traumatic Brain Injuries

Trust Us to Help You Treat Your Health Conditions

The name of the condition that creates the pain that plagues so many! You’d be hard-pressed to find a human being that has not experienced pain and inflammation in their joints. For some, the path was simple abuse and/or over-use without adequate care/time allowed for healing and repair. For others, it was a more metabolically mediated process where they were either chronically dehydrated or mineral imbalanced, or for many it happened through the process of consuming foods that triggered subclinical (sterile) inflammation.

Regardless of the initiating event, this process involves the development of excessive tension within an area of the joint cartilage. This biomechanical stress produces structural and biochemical changes within the cartilage, which result in the formation of pro-inflammatory chemical reactions. This leads to the destruction of cartilage and joint fluid. As cartilage and joint fluid break down, the joint loses its ability to absorb shock and more stress is applied to the adjacent bone. The surrounding bone degenerates and over-proliferates, forming spurs.

All of these metabolic and structural pathways cause a common state of inflammation that muscles respond to unfavorably and chronic muscle tension results. This leads to a reduction in the normal range of motion and the joints fail to hydrate adequately through regular daily movement. This reduction in motion results in joint inflammation and varying degrees of degeneration which we call osteoarthritis.

Interestingly, when the food sensitivity mediated inflammatory process is allowed to continue unabated there is a strong probability of the arthritic process to turn into an autoimmune driven process. The autoimmune inflammatory process of joint inflammation includes rheumatoid arthritis (RA), and juvenile rheumatoid arthritis (JRA). In the functional medicine realm, we address these conditions by targeting inflammatory processes at their core in a uniform fashion.

This Process Starts in the Gut

Over half of the body’s immune system is localized around the gastrointestinal tract. This means that anything that over—activates the intestinal immune system can result in a contribution to the arthritic process via cytokine formation. Some of the things that can activate the intestinal immune system include parasites, bacteria, viruses, fungi, toxins, and food antigens. A food antigen is a particle of food that is recognized by the immune system as a foreign invader. Although an individual can react to any food, some of the most common food triggers are wheat, dairy, citrus, yeast, nightshades, histamines, and shellfish.

Getting the repair process going functionally requires first stopping the inflammation and secondarily reading the environment where the joint can repair. Joint repair is influenced by numerous factors, including pH balance. Synthesis of new connective tissue is markedly impaired by excess tissue acidity. Acids are formed in response to consuming excess animal protein, grains, sugar, coffee, and alcohol. Stress, toxicity, and deficiencies of many nutrients also increase body acid. Aerobic exercise, hydration, and consumption of vegetables help neutralize acid.

Several other supplements are often helpful in cases of osteoarthritis. Niacinamide is one of these. High and frequent doses of niacinamide have been found to be very effective. Niacinamide can ease pain and stiffness, resulting in measurable increases in a joint’s range of motion as long as supplementation is continued and particularly so when inflammatory triggers are avoided.

There is so much that can be done for arthritic pain and even for the actual joint degeneration. The Functional Medicine model provides solutions for patients regardless of the pathways that brought them to their state of inflammation. The patient just needs to be willing to take an active role in their care beyond swallowing a pill to mask their pain.

Freedom From Fibromyalgia

Pain is generally the most prominent symptom of fibromyalgia. The condition often affects the entire body, although it may start in one area—such as the neck and shoulders—and spread to other areas over a period of time. The name, fibromyalgia, means: pain in the muscles and the fibrous connective tissues (the tissues and tendons), and is a form of generalized muscular pain and fatigue.

You (or someone you know) may be experiencing moderate or severe fatigue with a lack of energy, decreased exercise endurance, or the kind of exhaustion that results from the flu or lack of sleep. Sometimes the fatigue is more of a problem than the pain.

Headaches, especially tension and migraine headaches, are common in fibromyalgia. Abdominal pain, bloating, alternating constipation, bladder spasms, and irritability may cause urinary urgency or frequency. Your skin and blood circulation can be sensitive to temperature changes, resulting in temporary changes in skin color. Have you suffered from any of these symptoms for a few months, a few years, or too many years to remember? Some people may suffer only minor symptoms, while others (perhaps you or a loved one) feel the debilitating effects of fibromyalgia and are kept from participating in their life, making even simple daily activities almost impossible.

Over the past couple of decades, I have tested the full range of therapies and treatments available to me, generally with short-lived improvement. I knew there had to be more, a means of evaluation and treatment that could help correctly identify the cause and ultimately resolve this condition. In my studies of Metabolic and brain-based therapies, I have found the answer. The answer to helping most fibromyalgia symptoms lays in optimizing our main fuel delivery system for oxygen combined with enhanced brain-based therapy.

I was introduced to this breakthrough concept in care from my friend and mentor Dr. Michael Johnson author of the alternative medicine best seller “What Do You Do When the Medications Don’t Work? A Non-Drug Treatment of Dizziness, Migraine Headaches, Fibromyalgia, and Other Chronic Conditions.” After studying with Dr. Johnson and implementing the neurometabolic brain-based therapies, we have finally been able to help people with fibromyalgia find lasting relief from their pain and many other chronic symptoms. You are most likely starting to wonder “what is the treatment going to be like?” I can tell you from personal experience and from watching patients and staff participate in the treatments, that they are beneficial and enjoyable. Treatments can include uni-lateral mechanoreceptor stimulation, simple crossbody exercises, eye exercises, heat, and other activities, all while utilizing appropriate levels of oxygen. These techniques coupled with a full neurological and nutritional workup have enabled us to help so many of our patients get rid of their chronic symptoms regardless of how long they have been experiencing them. Imagine how different your life would be if you were free from the burdens that your fibromyalgia causes you.

You can learn more about this unique, corrective approach to fibromyalgia by viewing the video above and by scheduling a consultation with Dr. Marquis. We look forward to helping you soon.

It is important following an automobile accident to get care as soon as possible to help stop the inflammatory cascade happening in the body and, many times, in the brain. There are many things we can do to help with post-auto-accident injury and inflammation, including chiropractic work, following anti-inflammatory nutritional protocols, and/or utilizing various therapies that we have in our office. The plan of care that Dr. Marquis will put into care will include both a physical and neurological assessment and will be specific to each individual’s needs.

Vertigo, or dizziness, affects millions of people around the world each year. In the majority of cases, vertigo is nothing more than a temporary inconvenience. However, in some cases, vertigo is a debilitating condition where day-to-day activities are kept to a minimum due to the effects of severe dizziness.

Through vestibular nystagmography (an exam procedure where we have you wear digital video goggles to record fine motor control of your eyes) we have observed that in many cases the area of dysfunction in the nervous system is the cerebellum. The cerebellum is in the posterior aspect (back part) of the brain and controls our coordinated movements.

There are specific neurological tests that we utilize to determine cerebellar function. Some of them include global coordination tests such as standing with your feet together and eyes closed, the test is positive if the patient sways back and forth and is unable to maintain a fixed position. Other tests include more fine motor skills such as touching the index finger to the nose with the eyes closed, walking heel to toe, moving the fingers rapidly as if playing the piano, or touching all of your fingers to your thumb as fast as possible. Optical tests including convergence, pursuits, and observation for anomalies like catch-up saccades and exophoric eye movement are evaluated. Oxygen concentration versus oxygen perfusion is evaluated. These tests and others are used to determine the function of the cerebellum. Repositioning tests such as Dix-Hallpike are used to evaluate for the need for canalith repositioning.

Treatment of cerebellar dysfunction may include:

Unilateral (one-sided) mechanoreceptor stimulation: Extremities (arms and legs), lumbar (low back), and cervical (neck) mechanoreceptors may be stimulated on one side only to fire muscle spindle cells (muscle receptors) and joint mechanoreceptors (joint receptors) into the same side cerebellum and opposite cerebral cortex (brain). Vibration may be used to fire the dorsal columns (back part of the spinal cord) to facilitate proper cerebellar output.

In cases of benign paroxysmal positional vertigo, repositioning techniques such as the Epley maneuver may be implemented. In many cases, we use a warm caloric. This procedure produces rapid results, often simply amazing results are achieved in a brief period of time due to the powerful impact this has on a dysfunctional cerebellum. The procedure is done with warm water (about 180 milliliters), which is administered in the ear to stimulate the ipsilateral (same side) cerebellum.

Eye Exercises: Eye exercises may be used to increase the frequency of firing to the cerebellum and frontal lobe of the brain.

Laser Therapy: Laser therapy may be used to promote nitric oxide and ATP, which provides an increased metabolic and healing rate. Laser helps increase activation to the brain and it often has immediate soothing and palliative effects as a result of the vasodilatation and increased circulation.

One or all of the procedures may be employed to restore the cerebellum and vestibular apparatus to its normal function.